14-year-old presented with pain in anterior knee over 5 months time without any history of direct trauma.
14-year-old presented with pain in anterior knee over 5 months time without any history of direct trauma. Pain is worse during sporting activities. On examination patient is tender over inferior part of patella and over patellar ligament. MRI scan was done to confirm the clinical diagnosis and to rule out other knee pathologies.
Sinding-Larsen and Johansson syndrome was described independently by Norwegian physician Christian Magnus Falsen Sinding-Larsen and Swedish surgeon Sven Christian Johansson. It is a type of osteochondrosis at the proximal attachment of patellar ligament, which is common in adolescents especially those who participate in jumping activities. The aetiology appears to be traction tendinitis with de novo calcification in the proximal attachment of patellar tendon [1]. This may be caused by repetitive microtrauma to the tendon at the insertion to lower pole of patella. Clinically, patients present with pain, swelling and tenderness over inferior pole of patella precipitated by overstretching or trauma. Although it’s mainly a clinical diagnosis, imaging modalities help in diagnosis. Plain x-ray imaging may show osseous fragmented appearance of lower pole of patella [1]. MRI findings include fragmentation of lower pole of patella, marrow oedema within the fragments, thickening of patellar tendon at its insertion and oedema of Hoffa’s fat pad. Although ultrasound imaging is also effective and can be used for periodic follow-up during the course of this condition, the advantage of MRI is that it allows ruling out other intra-articular derangements [2-4]. As it is self-limiting condition, treatment involves conservative methods. The condition can last from three to twelve months [1]. Restricting activities such as jumping, kneeling, squatting, stair climbing and running should be avoided during the course of this condition. In severe cases brief period of immobilisation of affected knee joint may be beneficial. In the end the fragmented lower pole of patella may get incorporated into patella.
Sinding-Larsen and Johansson Syndrome
Based on the provided MRI images of the knee, the following observations are noted:
• There is a lobulated or fragment-like signal change at the inferior pole of the patella, with local signs of calcification or bony fragments, suggesting an abnormal bony appearance of the patellar inferior pole.
• Thickening and signal enhancement of the patellar tendon near its patellar attachment, suggesting stress-related changes or inflammation at the tendon insertion site.
• Localized edema signal in the Hoffa’s fat pad, indicating local soft tissue irritation or inflammatory changes.
• No obvious fracture lines or significant cartilage surface destruction are seen, and the joint space is generally preserved.
These changes are consistent with bone and soft tissue alterations caused by repeated traction at the inferior pole of the patella.
Considering the patient’s age (14-year-old adolescent), recurrent anterior knee pain, and MRI showing fragment-like changes at the inferior pole of the patella with soft tissue edema at the patellar tendon attachment, the most consistent clinical imaging diagnosis is Sinding-Larsen-Johansson Syndrome (traction apophysitis at the patellar inferior pole).
Disclaimer: This report is intended for academic and clinical reference only and should not substitute for an in-person consultation or professional medical advice. If you have any questions or if symptoms worsen, please see a physician promptly.
Sinding-Larsen and Johansson Syndrome