Lateral cord plantar fasciopathy

Clinical Cases 30.09.2019
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Section: Musculoskeletal system
Case Type: Clinical Cases
Patient: 40 years, female
Authors: Gheysens Gerald1, Mathieu Lefere2
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Details
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AI Report

Clinical History

A 40-year-old female patient presented to the radiology department with increasing, a-traumatic, lateral foot pain since three weeks.

Imaging Findings

X-ray of the foot was normal. On ultrasound, the lateral cord of the plantar fascia showed thickening, hypoechogenicity and loss of its fibrillar echotexture at the insertion on the base of the fifth metatarsal bone (Fig. 1). No hypervascularity was noted. The peroneus brevis tendon appeared normal (Fig. 2). No aberrant soft-tissue structure was found. Furthermore, the origin of the plantar fascia near the calcaneum was also hypoechoic and thickened in comparison to the contralateral side. The diagnosis of lateral cord plantar fasciopathy (LCPF) in addition to central cord plantar fasciopathy (CCPF) was made.

Discussion

Plantar fasciopathy typically causes heel pain or plantar foot pain. However, it can also cause lateral foot pain. Anatomically, the plantar fascia is comprised of a central cord and a lesser known yet clinically relevant lateral cord [1]. The central cord runs from the medial tubercle of the calcaneum and divides into 5 diverging bands, inserting onto the plantar plates of the metatarsophalangeal joints. Plantar fasciopathy most commonly involves the proximal third of this central cord. The lateral cord originates on the lateral calcaneal tubercle in close relation to the abductor digiti minimi muscle and inserts onto the plantar and lateral aspect of the base of the fifth metatarsal bone (Fig. 3). It can also be subject to fasciopathy, typically distally [2]. Moreover, CCPF can provoke LCPF by changing the gait of the patient, as was seen in this case. The sonographic features of LCPF are similar to other enthesopathies or tendinopathies. Generalised or focal hypoechoic thickening and loss of the normal fibrillar echo texture are the most frequent findings. Accepted standards for the normal thickness do currently not exist, obliging comparison to the contralateral side [3]. Cortical irregularity of the fifth metatarsal base and hypervascularity are possible, additional findings. The lateral cord of the plantar fascia inserts to the plantar and lateral aspect of the fifth metatarsal bone and the distal fibers widen into a broad attachment. On the other hand, the peroneus brevis tendon inserts to the dorsal and more distal aspect of the fifth metatarsal base with a small footprint [3]. Therefore, ultrasound can readily differentiate between LCPF and tendinopathy of the distal peroneus brevis tendon, the former being the more common cause of a-traumatic lateral foot pain. Written informed patient consent for publication has been obtained.

Differential Diagnosis List

Lateral cord plantar fasciopathy
Peroneus brevis tendinopathy
Fifth metatarsal stress fracture
Os vesalianum syndrome
Baxter neuropathy

Final Diagnosis

Lateral cord plantar fasciopathy

Figures

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Ultrasound shows thickening of the LCPF insertion on the symptomatic side (between calipers in a), compared to the contralate

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The peroneus brevis tendon (white arrows) had a normal echographic appearance. It inserts more superiorly and distally on the

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Anatomy of the lateral cord of the plantar fascia (white arrows), peroneus brevis tendon (yellow arrowheads) and abductor dig