Heel pain.
The patient presented with heel pain, which had increased in intensity over the previous few weeks and was especially severe in the morning. Sagittal T1-weighted MRI showed an increase in signal intensity and thickening in the proximal plantar fascia (Fig. 1). A sagittal T2-weighted FSE image showed oedema of the neighbouring soft tissues (Fig. 2). On a coronal STIR image bone marrow oedema centred on the origin of the plantar fascia was seen.
Repetitive trauma and mechanical stress are the most frequent causes of plantar fasciitis. Microruptures and inflammatory changes of the fascia and perifascial tissues result. Plantar fasciitis is common in runners and obese people (1,2). Inflammations of the plantar fascia may cause heel pain even without preceding trauma (1). Pain is perceived at the origin of the fascia. It is enhanced by dorsiflexion of the toes and it is more severe in the morning.
The lateral radiograph often shows a spur. This finding is unspecific however, because it is found in 25% of the symptom-free population. Scintigraphy may show increased uptake in the calcaneal region indicating a periostal inflammation (1). MR imaging is able to differentiate plantar fasciitis from other causes of heel pain, e.g. ruptures of the plantar fascia (1,2). On sagittal or coronal MR images the physiological plantar fascia is a thin, signal-free bandlike structure that extends anteriorly from the ventral undersurface of the tuber calcanei. The normal thickness of the plantar fascia is 3.22mm +/- 0.44mm and it flares at its calcaneal origin. Inflammatory changes result in thickening of the fascia and a signal increase in all sequences (intermediate signal on PD and T1-weighted images, high signal on T2-weighted images), These changes are pronounced in the proximal part of the fascia. Oedema may be seen in the subcutaneous fat and deep tissues, as well as bone marrow oedema centred on the origin of the fascia. The thickening of the fascia is often fusiform, which differs from plantar fibromatosis with its more focal, nodular thickening. In cases of rupture discontinuity of the fascia, oedema and haemorrhage can be seen.
Therapy is initially conservative, including orthesis and administration of non-steroidal antirheumatics. In more severe cases local injection of steroids and even resection of the fascia may be necessary.
Plantar fasciitis
Based on the provided MRI images, marked thickening of the plantar fascia (primarily at its origin along the medial calcaneal border) is observed. On T1-weighted sequences, the fascial signal is mildly increased, and on PD or T2-weighted sequences, a hyperintense signal can be seen, suggesting local edema and inflammation. The fascia thickening appears fusiform, especially near the calcaneal attachment, with mild surrounding soft tissue edema. No significant fascial discontinuity is noted, and there is no clear evidence of a large-scale tear. A mild abnormal signal in the adjacent calcaneal bone marrow may indicate bone marrow edema or inflammatory changes.
The patient is a 62-year-old female presenting with hindfoot pain, which aligns with the common presentation of plantar fasciitis. Imaging findings demonstrating significant thickening of the plantar fascia near the calcaneal attachment, altered signal, and possible calcaneal bone marrow edema strongly support a diagnosis of plantar fasciitis.
This report is based on the available images and clinical information, providing general reference recommendations only. It does not replace an in-person diagnosis or an individualized treatment plan by a professional physician. If you have any concerns or if symptoms worsen, please seek further evaluation at a hospital without delay.
Plantar fasciitis